![]() ![]() ![]() I still think the only thing he's going to be concerned with is whether or not I've started the Mediterranean diet to preserve my hearts health years down the road. He scoffed when my original monitor only had 7 PVCs, but now I'm showing up with 40k of them + thousands of PACs. I see my doctor on Tuesday regarding the results. Stress in my life is a constant right now. I'm a 39 year old male, 6 foot, 240 pounds (yes, I know I need to lose weight) with 4 children, and I do suffer from severe anxiety. If you have high blood pressure, you may need to take medication to help manage it. There was also one occurrence of "Sinus Rythm with IVCD." My doctor didn't even acknowledge it on the report. This is a device that emits electricity to help your heart maintain a consistent rhythm. The results were 2% burden of PVCs (I had over 40,000 of them lol) and a 1% burden of PACs, which I wasn't aware I was getting. So, last month, I demanded a 30-day heart monitor because my heart doesn't feel right, and my doctor gave in to my wish. I'm not sold that my chest pain is from this, and I've continued to get PVCs. I ended up seeing a GI doc, and I've had two endoscopys and I was diagnosed with Eosinophilic esophagitis, and I have a very small hiatal hernia. My doctor said nothing is wrong with my heart, even though I'm constantly getting severe chest pain that goes into my back, and I get PVCs and can feel most of them. The doctor said it's not causing any of my symptoms, but if I don't change my diet, it'll impact my heart within the next 10-20 years). CT Scan with Contrast - normal (but they saw the smallest amount of plaque possible forming in the left and right coronary arteries. 30 chest xrays (I went to the ER and Urgent care 20 times in the last two years for chest pain, which is why I had so many chest xrays) - normal. The result was 7 PVCs, that's it! But, this was an event monitor, so it wasn't constantly recording.Īt the time, my cardiologist was not concerned about my heart, but I insisted on follow-ups. In March of 2021, I saw a cardiologist, and he put me on a 10-day event monitor. These findings suggest that a prolongation of the QT interval may be a marker for the therapeutic antiarrhythmic effect of amiodarone.I started getting shortness of breath and chest pain in 2020. The levels of amiodarone (2.5 versus 3.2 micrograms/ml) and its metabolite (desethylamiodarone) were not significantly different between the living patients and those who died suddenly. No difference was observed in the percent change in QRS interval between the two groups. A significant difference in percent QT prolongation was seen between the latter patients and those who died suddenly (p less than 0.005). The addition of plasma sodium, age and NYHA class had no added benefit on the predictive power of the model. A model based on multivariate analysis showed that IVCD, MVO2 and left ventricular ejection fraction (LVEF) were the best predictors of mortality. Marked prolongation in the QT interval was present in patients who remained alive with amiodarone therapy. On bivariate analysis, IVCD and MVO2 were better predictors when combined together. Using a two-way analysis of variance, the percent change in QT, QTc, JT and JTc intervals before and after amiodarone therapy was analyzed. Abnormal septal motion (commonly referred to as septal bounce) is a common echocardiographic finding that occurs with several conditions, including the following: mitral stenosis, left bundle branch block, pericardial syndromes and severe pulmonary hypertension. Ten died: six suddenly, three of non-cardiac causes and one of congestive heart failure. Twenty-three are alive after a mean follow-up period of 12 +/- 7 months. There were 30 men and 3 women (mean age 52 +/- 10 years). The electrocardiogram and amiodarone levels were evaluated in 33 patients who presented with cardiac arrest and symptomatic ventricular tachycardia in whom no other antiarrhythmic agent was found effective in preventing induction of ventricular tachycardia during electrophysiologic studies. Prolongation of the QT interval in patients dying suddenly was compared with that in patients who remained alive to determine whether a difference existed between these two groups. Amiodarone is an antiarrhythmic agent known to cause prolongation of action potential duration which is reflected in the electrocardiogram as a prolongation of the QT interval.
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